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Figure c10/f001
Figure 10.1
Wound inflammatory cells during the first 2 weeks of wound healing showing the stages of acute wound healing and the inflammatory infiltrate present i...
Figure c10/f005
Figure 10.5
Wound contraction to initiate scarring. Macrophage‐derived platelet‐derived growth factor (PDGF), transforming growth factor‐β (TGF‐β) and fibroblast ...
Figure c10/f009
Figure 10.9
Examples of biological skin substitutes.
Figure c10/f002
Figure 10.2
Matrix metalloproteinases (MMPs) and tissue inhibitor of matrix metalloproteinases (TIMPs) in wound healing showing the expression and cellular source...
Figure c10/f006
Figure 10.6
Model of how secreted heat shock protein 90α (HSP‐90α) promotes wound repair. Injury to the skin triggers the release of transforming growth factor β ...
Figure c10/f003
Figure 10.3
Mechanisms involved in matrix degradation during keratinocyte migration. Physiological control of pericellular proteolysis occurs primarily through th...
Figure c10/f007
Figure 10.7
Better and faster skin wound healing is achieved by the combined action of AMD3100 and tacrolimus. AMD3100 interferes with the interaction of the CXC ...
Figure c10/f004
Figure 10.4
Molecular and cellular factors required for angiogenesis in order to promote endothelial cell migration, proliferation and tubule formation. After wou...
Figure c10/f008
Figure 10.8
Keratinocyte grafting. (a) Sheet of cultured keratinocytes for grafting (b) Ultrastructure of keratinocyte graft. Scale bar 1 μm. (Courtesy of Profes...